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Crit Rev Toxicol

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Increased cellular protein S -glutathionylation sensitized hepatocytes to TNF hepatotoxicity Increased cellular glutathionylated protein levels in response to GSSG accumulation prompted us to examine the effects of cellular protein S -glutathionylation on TNF-induced cell death

The resistant mutants rbohF and rbohD/F of Arabidopsis exhibited the induction of GSH form deposition and summary glutathione (GSH+GSSG pool) changed the activity of apoplastic GGT (-glutamyl transferase) in the apoplast, with the active rerouting of GSSG from the cell wall to the symplast during TuMV infection ( Figure 2 )