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GIP perhaps makes a greater contribution to the physiological insulin response to meals than GLP-1, but therapeutic development to date has focused on GLP-1, as the insulinotropic effect of GIP is markedly attenuated in Type 2 diabetes (T2D)

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In addition, novel studies demonstrate that GLP-1 inhibition may mediate clozapine-induced weight gain and metabolic dysregulation, suggesting that GLP-1 agonists could reverse both (58)

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