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Mester, S

Interference with the pentose phosphate pathway by directly targeting G6PD using the antiinflammatory compounds dimethyl fumarate or parthenolide leads to complete depletion of GSH and concentration-dependent eryptosis [113]

Consequently, mTORC1-deficient T cells cannot differentiate into Th1 or Th17 cells, and mTORC2-deficient T cells lack the ability of Th2 differentiation [66, 67]