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Persistent symptoms may reflect: Stored toxin burden Ongoing immune dysregulation Mitochondrial stress Autonomic nervous system instability Impaired elimination capacity Because mold-related illness involves immune and metabolic disruption not simply toxin presence recovery requires stabilization before aggressive detoxification is considered

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American Heart Journal

In parallel, GLP-1 receptor agonists have demonstrated neuroprotective actions beyond their established metabolic benefits, offering a promising avenue for cognitive preservation in at-risk individuals

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