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Pretreatment with Dula protected against AA-induced UC by decreasing colon mass index, colon weight and weight/length ratio, colonic macroscopic damage, and Dula also showed the ability to reduce oxidative stress damage via decreasing the lipid peroxidation end product, MDA, and restoring the decreased GSH, SOD, and TAC levels

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Moreover, although one can expect that elevated homocysteine levels result from the vascular disease per se (Kuller and Evans, 1998), the cardiovascular risk is gradually related to plasma homocysteine in many studies (Hopkins et al, 1995

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