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They are thought to be associated with activation of microglia, infiltrating leukocytes, proteins, and/or lipid exudates, resulting from the breakdown of the inner BRB and reflecting an underlying inflammatory process

Preclinical studies have demonstrated that TMAO can induce apoptosis in various target cells, such as human aortic vascular smooth muscle cells, 158 pancreatic acinous cells, renal tubular epithelial cells, 159 human aortic endothelial cells, 160,161 and neurons, 119 contributing to the development and progression of cardiovascular, renal, neurological, and pancreatic diseases

In a genetic preclinical study, embryonic GIPR knockout mice fed a high-fat diet were protected from obesity, supporting the role of GIPR antagonism as a method to promote weight loss and prevent weight gain (67)

doi: 10.1136/thorax.57.11.958

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