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560 Presenilin mutations in patients with AD may contribute to neurodegeneration by downregulating GPX4 and promoting ferroptosis

Pharmacological modulation of mitochondrial pathways presents significant challenges

Individuals with one or two copies of the genetic variation and low activity of the MTHFR enzyme may present with elevated homocysteine levels, which have been associated with inflammation, heart disease, pregnancy complications, and even miscarriages

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