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Complementing these findings, Mendelian randomization analyses using genome-wide association data suggest potential causal links between specific gut microbial taxa and PD risk, with effects partly mediated through inflammatory cytokines and metabolic intermediates such as tryptophan [196]

To this end, we performed bilateral injection of an AAV-DIO-hM4Di-EGFP virus into the PVN and implanted cannulas to locally perfuse CNO into the DMV of VgluT2-Cre mice (Fig

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They tend to reduce excessive inflammatory signaling without eliminating protective immunity

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