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Menard C, Pfau ML, Hodes GE, Kana V, Wang VX, Bouchard S, et al

Conversely, following hypoxia or loss of mitochondrial membrane potential, the inactivation or reduction of tyrosine kinase activity through the activation of PGAM5 will contribute to the downregulation of the phosphorylated FUNDC1 level

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Upon reaching 80% confluence, the cells were stimulated with 100 ng mL 1 LPS for 12 h to induce inflammation, which was confirmed by the overexpression of vWF on the inflamed HUVECs