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Loss of FeS cluster stability, often observed under oxidative stress, leads to the release of free iron into the mitochondrial matrix, exacerbating Fenton chemistry and fueling ferroptosis ( These findings highlight the pivotal role of mitochondrial dysfunction in ferroptosis-driven fibrosis

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Previous studies have established that Sororin recruitment partially relies on SMC3 acetylation (Lafont et al, 2010

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