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A bioinformatics analysis showed that ferroptosis was involved in the significantly enriched biological processes in the diabetic tubulointerstitium[158]

doi: 10.1016/j.jhep.2023.03.017 2

Specifically, by utilizing a transgenic mouse model where a dominant negative IGF-I receptor is expressed specifically in skeletal muscle (MKR), it has been shown that increased mechanical load can induce muscle hypertrophy and activate Akt and p70S6K independently of a functional IGF-I receptor, implying that IGF-I may not be a limiting factor in the induction of hypertrophy with muscle overloading 61