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Mechanistically, BPC-157 promotes fibroblast activity, modulates the nitric oxide system, enhances angiogenesis through vascular-endothelial growth factor, downregulates proinflammatory cytokines, and stimulates collagen remodeling (Figure 1)

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Rapamycin addresses cellular senescence in the follicle microenvironment by inhibiting mTOR, while GHK-Cu works on Wnt signaling, inflammation, and androgen receptor sensitivity