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It has been shown that HSYA was able to significantly protect the liver from oxidative stress by increasing the activities of antioxidant enzymes, upregulating the expression of PPAR- and MMP-2, and downregulating the expression of TGF-1 and tissue inhibitor of metalloproteinases-1 (TIMP-1), and reducing -SMA level, which indicating that PPAR-TGF-1 would be a signal pathways for HSYA to reduce liver fibrosis induced by oxidative stress (Wang et al., 2013)

It is documented that neurons express G-CSF receptors, and exogenous G-CSF promotes peripheral nerve regeneration and function significantly [56]

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[PubMed] [Google Scholar] 239.Andersson I., Gronberg A., Slinde F., Bosaeus I., Larsson S