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PMCID: PMC3296184

Results Absence of promoter proximal NRF2 at NRF2 signature genes To identify critical pathways downstream of NRF2 activation in cancer, we sought to define a core set of genes and other features associated with NRF2-dependent growth across an expansive panel of cancer cell lines

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Macrophage studies demonstrate that GHK-Cu administration shifts these cells from a pro-inflammatory M1 phenotype to a tissue-repairing M2 phenotype, increasing their production of growth factors including vascular endothelial growth factor (VEGF) and platelet-derived growth factor (PDGF) by 40-60%