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Thus, we have demonstrated that GSH-targeted therapy selectively kills PC cells in an oxidative stress-dependent manner and that the differences in basal ROS level and antioxidant scavenging capacity induced by stratified expression of VRK2 can well account for the heterogeneous sensitivity to GSH inhibition

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As women increasingly delay their first childbearing [7,8,9], there is a warranted need to better understand the mechanisms linking aging with the decline in fecundity and other reproductive challenges