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Therefore, inhibiting GSH in SWI/SNF-deficient cancer cells using eprenetapopt (APR-246) deals another blow by further decreasing basal GSH levels, thereby increasing basal ROS levels, which trigger apoptosis (Fig

When mitochondrial energy falls and reactive oxygen species rise, the effects may include cognitive slowing, poor stress tolerance, sensory overload, sleep disturbance, irritability, impaired impulse control, psychosis vulnerability, fatigue and reduced rehabilitation capacity

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