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Indeed remittance from diabetes experienced by RYGB or SG patients, has been attributed to the elevation of circulating bile acids (37, 38, 357), warranting further investigation, especially the development of gut-restricted TGR5 agonists (358)

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Deletion of both Rab-GTPaseactivating proteins TBC14KO and TBC1D4 in mice eliminates insulin-and AICAR-stimulated glucose transport