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It plays a central role in protecting cells from oxidative damage by neutralising free radicals, and is essential for mitochondrial function and cellular redox balance

These findings underscore DSIPs potential as a therapeutic agent for conditions involving mitochondrial dysfunction and oxidative stress, such as cerebral ischemia [1]

Experiments were performed in accordance with the Guidelines by the NIH Office of Laboratory Animal Welfare regarding the care and use of animals for experimental procedures, and following the recommendations stated in the ARRIVE guidelines ( The experiments were approved by the UTHealth-Houston Animal Welfare Committee (AWC) under animal protocol number 21-0065

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