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Abstract Hypoxic-ischemic brain damage (HIBD) is the main cause of perinatal mortality and neurologic complications in neonates, but it remains difficult to cure due to scarce treatments and complex molecular mechanisms remaining incompletely explained

10.1002/psc.782 13 BarlowN.VangaS

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the finding was corroborated in a transgenic model of ALS where rats or mice express the mutant Cu 2+ /Zn 2+ -dependent superoxide dismutase (SOD1G93A) originating from a form of human familial ALS

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