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Elevation of IGF-1 levels to upper normal or mildly supraphysiological ranges has raised theoretical concerns about cancer risk, as IGF-1 can promote cell proliferation and some epidemiological data link high IGF-1 with increased cancer risk (particularly colorectal, prostate, and breast cancers, though data is mixed)

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This approach targets fat loss through multiple mechanisms without relying on appetite suppression

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We thus tried to investigate the potential for VRK2 phosphorylating Sec24C and found that the phosphorylation level of Sec24C was attenuated in VRK2-KO cells but elevated when restoring the expression of VRK2 (Fig