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A smaller pilot study also revealed that cerebral glucose delivery was determined by plasma glucose concentration and cerebral blood flow, with injured brains exhibiting less glucose uptake and utilization around lesions but increased utilization at distal hyperglycolytic sites in the brain [32]

Some studies revealed that because of high concentration in plasma, UA can act as a pro-oxidant within the cell by scavenging and creating ROS or it can act as an antioxidant, which mainly occurs in plasma [122,123,124]