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The activation of the post-receptor signaling cascade induced by GLP-1 and GLP-1 receptor agonists and the consequent modulation of insulin signaling pathway would reduce storage of hepatocyte triglyceride and consequently improve hepatic steatosis [83]
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Research data suggests that approximately 70-80% of adverse effects are classified as mild, with subjects able to continue their research participation with minimal intervention [2]
Regarding pharmacodynamics, TZP appears as a biased GLP-1R-GIPR co-agonist, because of its positive effects on cAMP generation over -arrestin recruitment, which is conversely stimulated by GLP-1R
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