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Genetic instability caused by persistent cancer cell oxidative stress will therefore lead to the increase of the malignant potential of the tumour, due to the inactivation of additional tumour suppressor genes in tumour cells, or via increasing expression of proto-oncogenes (252, 253)

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Unlike GLP-1R, N291N298 segment of ECL2 in the ligand-free GCGR stretches toward the TMD core to partially occupy the orthosteric pocket, which overlaps with the N-terminal segment (T5 P S8 P ) of GCG (Fig