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Strategies involving pharmacological or genetic inhibition of PGE2 production and signaling, either alone or in combination with PD-1 blockade, have been shown to enhance CD8 + T cell effector function during chronic LCMV infection [71]

In circulation, the vast majority of IGF-1 exists bound to one of the IGFBPs, with IGFBP-3 accounting for approximately 80% of all binding