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Therefore, a possible mechanism proposed by the authors is that the activation of the farnesoid X receptor (FXR) by exendin-4 stimulates the expression of FGF15 in the small intestine and inhibits the expression of cytochrome P450 family 7 subfamily A member 1 (Cyp7a1) in the liver through fibroblast growth factor receptor 4 (FGFR4), thereby reducing fecal bile acid excretion and liver bile acid synthesis [75]

ALT and AST enzyme levels normalize in many patients

Furthermore, infiltrating macrophages, cytokine production and inflammatory pathways are systemically increased during aging (inflammaging) also at the skeletal muscle level with a concomitant alteration of redox-mediated signaling pathways and muscle loss [92,93,94]

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