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The KEAP1 protein detects oxidative stress by going through a conformational change that prevents association with NRF2, leading to NRF2 accumulation and translocation to the nucleus, which then induces transcription through antioxidant response elements found in a broad set of genes involved in redox homeostasis and correcting oxidative damage
It would appear that there is an optimal level of IGF-1 that lends itself to peak metabolic health, with a deviation towards deficiency or excess leading more towards metabolic dysregulation
Quni S, Zhang Y, Liu L, Liu M, Zhang L, You J, et al
It should not be attempted in non-medical settings
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