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During pathogen attack, lignin precursors undergo extensive cross-linking, via H 2 O 2 -mediated pathways to reinforce the cell wall with lignin (Higuchi, 2006)

While the clinical research surrounding N-Acetyl-L-Cysteine is highly encouraging, it is important to maintain a realistic outlook

A surrogate result is weaker evidence than an outcome result

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One possible explanation is regional variation in Cre recombination efficiency under chronic metabolic stress