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These effects promote angiogenesis, fibroblast activity, and neuromuscular stabilization, particularly in poorly vascularized tissues such as tendons and myotendinous junctions

In the early 2000s, the Australian company Metabolic Pharmaceuticals ran a series of clinical trials to assess AOD 9604's effectiveness in humans

Both are studied in tissue repair contexts but via distinct mechanisms

DOI:10.3389/fphar.2021.627533

[10] The suggestion that it preserves physiological feedback regulation is a reasonable mechanistic concept, but clinical outcome superiority over licensed recombinant human GH (somatropin) has not been established