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Physiological melanin hyperpigmentation is the result of increased melanin biosynthesis by melanocytes in the basal cell layer of the oral epithelium, but pathological melanin pigmentation may be the result of increased number of normal melanocytes or atypical melanocytes, of increased melanogenic activity of normal or atypical melanocytes, or of both

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Furthermore, EcN signals through TLR-2 and TLR-4 dependent mechanisms to engage host immune responses, regulating the secretion of various immune mediators and promoting immune homeostasis [53]