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Whether these safety signals translate equivalently to MC4R -deficient patients - who are predominantly children and adolescents with a distinct metabolic phenotype - remains unknown

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These neurons send widespread projections to brain regions that highly express GLP-1Rs [66, 69], where administration of GLP-1R agonists reduces food intake and GLP-1R antagonists increase food intake such as the bed nucleus of the stria terminalis (BNST) as a notable example [70] (Fig

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