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We're going to break this down from a practical, lab-focused perspective

In summary, GSH enables antibiotic inactivation by serving as a substrate for detoxifying enzymes, such as FosA, and by modulating the expression or stability of antibiotic-degrading enzymes, including the downregulation of NDM-1

Acta Physiologica Hungarica

Furthermore, lnc-MRGPRF-6:1 showed high expression in monocyte-derived macrophages from CAD patients and exhibited negative correlation with GPX4 expression (60)