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Abstract Background Coronary artery disease (CAD) persists as a major global health burden, contributing significantly to both morbidity and mortality rates worldwide, mostly attributable to atherosclerosis and oxidative stress

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These include persistence of SARS-CoV-2 reservoirs, presence of microthrombi, induction of autoantibodies, hyperreactive immune activation, reactivation of other latent viral coinfections such as Epstein-Barr Virus (EBV), mitochondrial dysfunction, and gut dysfunction/dysbiosis (11)