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Notably, key transcription factors (e.g., Nrf2 and HIF-1) act as critical regulatory checkpoints within this network: Nrf2 inhibits ferroptosis and inflammation by upregulating GPX4 and antioxidant enzymes, while HIF-1 exerts a context-dependent dual role, acutely mitigating ferroptosis via Nrf2 activation but promoting fibrosis and sustained ferroptosis when chronically stabilized

Gann, J