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Its abnormal glycosylation modification (such as lack of N-glycosylation or insufficient O-glycosylation) can lead to hENT1 membrane localization disorder (endoplasmic reticulum degradation or intracellular retention) or decreased stability (accelerated protease degradation) (Chakraborty et al., 2018), thereby reducing gemcitabine influx and activation

Immune-supporting and immunomodulatory properties of new pro-glutathione (GSH) molecules

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Despite decades of research on ASD, identification of the causes of and treatments for ASD remain limited

(60) showed that the ethanolic extracts of H