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Examining direct GLP-1R-mediated cardiomyocyte protection through PI3K-Akt and AMPK pro-survival pathway activation in cardiac ischaemia-reperfusion models, endothelial GLP-1R-mediated anti-inflammatory and anti-atherosclerotic biology, the relative contributions of direct cardiac GLP-1R signalling versus systemic metabolic improvements through weight and glucose reduction to cardiovascular outcome benefits, and the mechanistic basis of GLP-1R-mediated plaque stabilisation, endothelial function improvement, and cardiac remodelling attenuation
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Data sources We performed a secondary analysis of EHR data obtained from three health systems participating in either the Helix Research Network and/or the Healthy Nevada Project
Most of the interest comes from researchers who are exploring the potential of semaglutide to address the inflammatory and metabolic aspects of fibromyalgia
