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[10] These extensions are best read as hypotheses the authors find worth pursuing, not as demonstrated systemic outcomes

NNMT inhibition addresses the deficit by preventing the enzyme (NNMT) that wastes the precursor from inside the cell

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Third, although we have previously reported that elevation of HO-1 induced lethal fibrosis in acute exacerbation of IPF triggered by COVID-19, it is necessary to perform a molecular pathological study regarding the in vivo dynamics of serum HO-1 elevation and pro-fibrotic cytokines such as tissue growth factor-1 43