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The main role of Xdh was to act as a catalyst for the conversion of hypoxanthine to xanthine, and ultimately to uric acid [81]

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This is theoretically sound but clinically unproven

As an upstream event of ferroptosis, autophagy activation can induce ferroptosis through different mechanisms, including NCOA4-mediated ferritin autophagy [95], RAB7A-mediated lipid autophagy [96], ARNTL-mediated biological clock protein autophagy [97], and BECN1-mediated System X c inhibition [64]

Addressing glutathione redox status in clinical samples by two-step alkylation with N-ethylmaleimide isotopologues