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Subsequent studies have shown that this phenomenon could be linked to a slowing of gastric emptying by GLP-1, as well as by the action of this hormone at the level of certain neural circuits of the brain involved in the suppression of appetite and the development of nausea

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It also reprograms CRC cell metabolism, enhancing glucose utilization and mitochondrial activity to support growth in hypoxic conditions

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Patients already on semaglutide for diabetes who want to treat MASH should talk to their provider to see if dose escalation is right for them, says A