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Description

Chronic synaptic insulin resistance after traumatic brain injury abolishes insulin protection from amyloid beta and tau oligomer-induced synaptic dysfunction

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5C, Supplemental Figs

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This varied ontogeny is reflected in distinct mutational profiles 7 , histological features and molecular phenotypes, resulting in complex classification schemes aimed at different clinical objectives 8

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Cryptococcus: from environmental saprophyte to global pathogen

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