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Smad activation-mediated TGF-1 inhibited Gln catabolism in dermal fibroblasts, thereby attenuating the expression of profibrotic markers in SSCs (86)

The area of these fibrinaloid microclots was found to be greater in ME/CFS samples in comparison to healthy controls (313)

Its pathogenicity is closely linked to specific adhesion-associated genes (e.g., fimA, sfaA, and focA) and aminoglycoside-resistance genes (aph(6)-Id, strAB), which facilitate both mucosal colonization and evasion of conventional antimicrobial therapy 134

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