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& Yamaguchi, Y

Laura Slattery, Specialist Registrar in Nephrology and General Internal Medicine, Royal College of Physicians, Dublin

Higher levels of corticosterone in ethanol-fed Aldh2 -/- mice were probably due to higher levels of acetaldehyde in these mice versus ethanol-fed WT mice because acetaldehyde can directly stimulate corticosterone release with 10 times more potency than ethanol.46-48 Importantly, we also demonstrated that acute alcohol drinking rapidly elevated serum levels of both cortisol and corticosterone in human subjects with much higher levels in those with ALDH2 deficiency