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Posted April 21, 2025 Reviewed by Monica Vilhauer Ph.D

While existing animal models, which replicate specific phenotypes by targeting individual organ systems, offer the closest approximation to the HFpEF pathology, they still fall short of fully recapitulating the human condition

Journal of Nanobiotechnology 19(1):118

A declining vitamin B6 status and a concurrent lower dietary intake of choline/betaine suggests a perfect storm leading to raised homocysteine and a higher inflammatory profile due in part to inadequate supply of methyl donors to the methionine cycle

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