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However, these residues are on the opposite face of the NTD relative to the expected interface of the core domain and therefore may form an interface with another, as yet unidentified, protein

Either by EBV primoinfection or infection by another pathogen that decreases CD4 T-cell responsiveness would increase the number of EBV-latent cells in different tissues, as EBV type I latent B cells are only controlled by EBNA-1-specific CD4 T cells (123125)

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