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We identified a subset of metabolites that distinctly characterize epilepsy-associated metabolic changes, including citric acid, glutamic acid, proline, 3-methyl-2-oxovaleric acid and glucose (Fig

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Nonetheless, despite these limitations, our study offers significant insights into the direct impact of metformin on intestinal glucose metabolism, highlighting the role of TXNIP downregulation and GLUT1 upregulation as a key mechanism of its glucose-lowering effect