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In the absence of MTAP, the substrate methyl thionyl adenosine (MTA) accumulates and acts as a selective inhibitor of PRMT5 to inhibit PRMT5 methylation activity, 237 whereas MAT2A can produce the PRMT5 substrate SAM, 238 allowing PRMT5 to exert its oncogenic effect
[PMID:33189300] Lau CT, Au DM, Wong KKY
2004 Nov;61(5):650-2
Additionally, the study indicated a potential decrease in the expression of urea cycle enzymes in animal models
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