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Simultaneously, shedding or mutation of the extracellular region of some membrane proteins leads to pseudotarget or target loss, resulting in targeting moieties that do not effectively recognize the target receptor

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The results in humans, however, were modest enough that it has not advanced to Phase 3 approval for any fat loss indication

More recently, however, an animal study based on a tandem stenosis mouse model showed the ability of SGLT2is to stabilize atherosclerotic plaques, with increased fibrosis, augmented collagen accumulation, and significant upregulation of the expression of vasculoprotective NADPH oxidase 4 [18]