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These include direct effects on GLP-1R + cells within joints, the heart, kidney, liver, and immune cells 14 and indirect actions through weight reduction and blood glucose control, as well as through interorgan communication to dampen inflammation via activation of GLP-1R in subsets of central nervous system neurons 15 (Figure 1B)

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Gastrointestinal research is heavily weighted toward BPC-157, reflecting its origin as a gastric-derived peptide

The expression of VE-cadherin (VE-Cad), an adherens junction protein and a key regulator of endothelial barrier function [77], was assessed in this model, showing cell elongation, which indicates the presence of a tight endothelial-like monolayer [43, 44]