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Further, hypoxia in the tumor microenvironment promotes lactate accumulation, extracellular acidosis, VEGF overexpression, and VEGFR activation, all of which are known drivers of immune cell tolerance and immunosuppressive status (Mendler et al., 2012

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This overexpression triggers significant oxidative stress, increasing lipid and protein oxidation levels, and ultimately exacerbates atrophy through the ubiquitin-proteasome pathway, likely stimulated by muscle-specific E3 ligases Atrogin-1/MAFbx and MuRF1

When it comes to anything going into your body, the source is not a detail