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Analysis of the genome sequence indicates that TGR is a single-copy gene (unpublished data) and no other activity is present in worms for Trx and GSH reduction [11]

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In pathophysiological situations, as occurs in Zucker obese rats, exendin-4 seems to act as a compensator for the variations in AMPK activity produced either by oscillations in glucose levels or by pathologies such as obesity or episodes of hyperinsulinemia

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