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Functional studies in receptor-deficient models further confirm that 3-adrenergic signaling is essential to its activity, positioning AOD 9604 as a tool for dissecting receptor-mediated lipid mobilization [1]

All participants were administered an identical intervention (1cm 3 of saline containing 200 mg GSH) using a syringe attached to a Mucosal Atomization Device supplied by Wolfe-Tory Medical (Salt Lake City, UT, USA)

Sections were blocked with a serum-free buffer to minimize nonspecific staining and incubated overnight with a purified polyclonal rabbit anti-human Humanin (HN) antibody at 7 g/mL

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